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  Indian J Med Microbiol
 

Figure 5: (a) Distribution and excretion of normal saline (0.9% NS) after intravenous administration. The high chloride load is above physiological limit and can contribute to hyperchloremic metabolic acidosis. (b) Distribution, metabolism and excretion of balanced salt solution (Ringers lactate and Plasmalyte A) with intact EGL layer. (c) Distribution and excretion of balanced salt solution (Ringers lactate and Plasmalyte A) with damaged EGL layer leading to edema formation. ECF – Extracellular fluid, RL – Ringer's Lactate, PL – Plasmalyte A, Cl- – Chloride, HCO3— – Bicarbonate

Figure 5: (a) Distribution and excretion of normal saline (0.9% NS) after intravenous administration. The high chloride load is above physiological limit and can contribute to hyperchloremic metabolic acidosis. (b) Distribution, metabolism and excretion of balanced salt solution (Ringers lactate and Plasmalyte A) with intact EGL layer. (c) Distribution and excretion of balanced salt solution (Ringers lactate and Plasmalyte A) with damaged EGL layer leading to edema formation. ECF – Extracellular fluid, RL – Ringer's Lactate, PL – Plasmalyte A, Cl- – Chloride, HCO3— – Bicarbonate